RR 3, Box 384
THE NUTRI-SPEC LETTER
Guy R. Schenker, D.C.
It may be a matter of life and death; it is certainly a matter of ...
VIBRANT LIFE VS FEELING LIKE DEATH.
It most directly involves your NUTRI-SPEC Glucogenic/Ketogenic and Sympathetic/Parasympathetic metabolic balance systems. It is the happy result of compliance with your NUTRI-SPEC Fundamental Diet. It is achieved only by those well enough informed to scorn the low saturated fat, low cholesterol diet advocated by the common wisdom of our day. It is celebrated most fully in those who laugh at the patently false omega 6 and omega 3 fatty acid propaganda so heavily hyped by todayís nutrition ďauthorities.Ē What is it? We are referring to one of the greatest treasures you offer your NUTRI-SPEC patients ...
Blood sugar, and blood insulin have a more immediate and more powerful impact on how we feel at a given moment than any other nutrition-related factor. Only ingestion of omega 6 and omega 3 fatty acids and their subsequent oxidation or conversion into prostaglandins can rival loss of glycemic control as a mechanism by which we ...
FEEL REAL ROTTEN, RIGHT NOW.
[In other words, if you feel suddenly yucky --- look to your last two, or maybe three meals. You will likely find that you either poisoned yourself with PUFAs, or, that one or more of those meals violated the rules of glycemic control.]
In response to any meal, there are seven factors that determine an individualís ability to maintain glycemic control:
Number 1 in the above list, consideration of each individualís metabolic imbalances, is of primary importance to you, the NUTRI-SPEC practitioner. Using objective testing procedures to discover and correct each of your patientís metabolic imbalances is what sets you apart from all other practitioners of clinical nutrition. You are special in understanding that your NUTRI-SPEC key concept of biological individuality is what determines ...
WHO WILL BECOME DIABETIC, WHEN, AND HOW.
You are special because your metabolic testing procedures give you the power to apply that key concept of biological individuality, so that you understand ...
WHO WILL BECOME HYPOGLYCEMIC, WHEN, AND HOW.
It is most fundamentally essential that you understand how Type I diabetes, Type II diabetes, and hypoglycemia fit into your NUTRI-SPEC metabolic balance paradigm. The various types of breakdown in glycemic control generally follow these patterns:
Of course, the causes and the effects of poor glycemic control are modified in any individual by the presence of Electrolyte, Anaerobic/Dysaerobic, or Acid/Alkaline Imbalances.
In last monthís Letter, we introduced you to the idea of the glucose/insulin tolerance test (GITT). Below is a graph of normal glucose-insulin curves, showing the response of a fasting person to a meal that is in accord with the NUTRI-SPEC Fundamental Diet.
You see fasting glucose around 85, that increases to about 130 in the first hour after the meal. From that peak of 130, the glucose drops over the next 2 hours to below 100, then more gradually back to the fasting level of 85. Meanwhile, the production of insulin is stimulated very rapidly as the blood sugar begins to rise immediately after the meal. As soon as the blood sugar begins to drop from its peak of 130, the insulin level drops even faster than does the blood sugar level, returning to the normal fasting level in less than 2 hours. (Remember, we are not showing a GITT curve in response to a test that involves ingestion of pure glucose. This is the normal glucose and insulin curve in response to a healthy meal with proper proportions of protein, fat, and carbohydrate.)
Let us consider now the consequences of ignoring the NUTRI-SPEC Fundamental Diet with respect to the proper proportions of protein, fat, and carbohydrate. Meals that are either excessively high in carbohydrate, or, that include carbs with a particularly high glycemic index, yield an exaggerated insulin response. The elevated insulin:
The clinical effects? --- Obesity, elevated triglycerides, and elevated cholesterol.
The high carbohydrate meals also (since they are relatively deficient in protein) cause a decreased production of glucagon. The combination of increased insulin and/or decreased glucagon causes a hypoglycemic reaction in most of us, and that reaction is exaggerated in those who tend to be glucogenic or parasympathetic. In those patients who tend to be ketogenic, however, the increased insulin does not precipitate a hypoglycemic reaction but instead, because of insulin resistance, is associated with a persistently high blood glucose. These ketogenic patients are those who succumb to the metabolic syndrome --- increased abdominal fat, diabetes, elevated triglycerides, elevated blood pressure, and cardiovascular disease.
Another consequence of the low glucagon produced in response to a high carb meal, is the increased conversion of arachidonic acid into nasty prostaglandins and leukotrienes. These cause the same symptoms --- allergies, headaches, arthritis, premenstrual symptoms, etc. --- as does the ingestion of PUFAs. With low glucagon there is also a lack of stimulus to the liver to release glycogen as glucose into the bloodstream, thus contributing to hypoglycemic reactions.
So, in summary, and referring to the graphs on the previous page, there is an increased insulin effect in response to high carbohydrate meals by glucogenic and parasympathetic patients. Those who are parasympathetic have an increased insulin output. Those who are glucogenic have increased insulin sensitivity, as well as probably a decrease in glucagon production. On the other hand, in your ketogenic patients and, to a certain extent in your sympathetic patients, the excess insulin produced in response to a high carbohydrate meal shows a decreased insulin effect, and a prolonged blood insulin level. Your sympathetic patients produce an insufficient quantity of insulin. Your ketogenic patients have insulin resistance, probably accompanied by increased glucagon production.
Is a high carbohydrate meal the only way to violate the NUTRI-SPEC Fundamental Diet? No, a person can also eat an excessively high protein meal. Excessive protein can be a problem for your sympathetic patients, and it is always a problem for your ketogenic patients. A high protein meal, like a high carb meal, increases insulin output, which is a problem for your ketogenic patients, causing them to store protein as fat, and, to decrease the release of stored fat. Since your ketogenic patients are insulin resistant, the damaging insulin circulates at chronically high levels. The high protein meal also stimulates the pancreatic release of excessive glucagon, which elevates the blood sugar of your sympathetic and ketogenic patients, many of whom are diabetic, or at least pre-diabetic.
The high protein, low carbohydrate diet can also throw your patients into ketosis. For those who tend to be glucogenic or parasympathetic ...
KETOSIS IS BLISS.
But for those who are ketogenic, ketosis can initiate changes in fat cells such that they are as much as ten times more active in storing fat. In sympathetic and ketogenic patients on a high protein diet, muscle mass can be catabolized for glucose to supply the brain.
You should now be able to answer the Quiz at the end of last monthís Letter with ease. Match the Graphs with Johnís dinner, Maryís dinner, Johnís breakfast, and Maryís breakfast.